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How the growth hormone axis is studied: GHRH analogues such as CJC-1295, sermorelin and tesamorelin, ghrelin-receptor agonists such as ipamorelin and the GHRPs, somatostatin, IGF-1 and kisspeptin.
How the three-tier Ras-RAF-MEK-ERK cascade works, why signal duration rather than signal presence decides cell fate, why a pathway inhibitor can switch the pathway on, and which research compounds genuinely have published ERK data.
The inhibitory half of the growth hormone axis: what somatostatin is, the five receptors it works through, why its three-minute half-life produced octreotide, and why the claim that GHRPs suppress it is not supported.
What each half of the CJC-1295 and ipamorelin pair is, where the synergy argument actually comes from, why no study has tested this combination, and how to verify two peptides in one vial.
Sermorelin and ipamorelin compared: a 29-residue fragment of a human hormone against a designed pentapeptide, their very different regulatory histories, and why no study has tested one against the other.
CJC-1295 with DAC explained: the Montreal-made GHRH analogue that binds albumin and what the 2006 human studies found.
Ghrelin explained: the 28-amino-acid stomach hormone found in 1999, why it needs a fatty-acid tag, how it drives hunger and GH release, and the GHRP link.
IGF-1 LR3 explained: the 83-amino-acid IGF-1 analogue built in Adelaide for cell culture, why it escapes binding proteins, what it's used for, and sourcing in Canada.
Growth hormone peptides explained: GHRH analogues vs ghrelin mimetics, how CJC-1295, ipamorelin, tesamorelin and GHRPs compare, and sourcing in Canada.
GHRP-6, GHRP-2 and hexarelin compared: the Bowers peptides that led to ghrelin, how their effects differ, approval status, and sourcing in Canada.
Kisspeptin explained: the gene named after Hershey's Kisses, its role as gatekeeper of puberty, what kisspeptin-10 studies show, and Canadian sourcing.